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您请求的操作仅限于组中的用户:==氧气治疗的风险== " ''通气抑制:“[[COPD(慢性阻塞性肺疾病)|COPD]]慢性二氧化碳(CO2)潴留患者存在缺氧呼吸驱动呼吸Kim V, Benditt JO, Wise RA, Sharafkhaneh A.慢性阻塞性肺疾病的氧气治疗。美国胸科学会会刊，2008;5(4): 513 - 8。Doi: 10.1513 / pats.200708 - 124等。< / ref >。当动脉张力升高到正常水平时，这些患者会失去维持通气所需的高碳水化合物刺激，导致通气不足Ergan B, Nava s。慢性阻塞性肺病,2017;14(3):351 - 366。doi: 10.1080 / 15412555.2017.1319918。< / ref >。“‘2。高压氧毒性:“长期高压氧2治疗可导致肺、视神经和[[脑干|中枢神经系统]]毒性Heyboer M, Sharma D, Santiago W, McCulloch N.高压氧治疗:副作用的定义和数量。” Advances in Wound Care, 2017; 6(6): 210–224. . '''3. Fire hazard:''' Oxygen enhance combustion of other fuels. Least level of supplemental oxygen (FiO2 greater than the 21% oxygen in ambient air) should be administratered during laser bronchoscopy to avoid intratracheal ignitionMillette BH, Athanassoglou V, Patel A. High flow nasal oxygen therapy in adult anaesthesia. Trends in Anaesthesia and Critical Care, 2018; 18: 29-33. '''4. Absorption atelectasis:''' Given only pure oxygen results in the collapse of the dependent part of the lungs as it quickly taken up from the alveoli. It is also a risk in general anaesthesia inductionHedenstierna G, Rothen HU. Atelectasis formation during anesthesia: causes and measures to prevent it. Jornal of clinical medicine and computing, 2000; 16(5-6):329-35. '''5. Retinopathy of prematurity (ROP):''' It usually occur in low birth weight, very premature infant. That is why in preterm infants, 50-80 mmHg PaO2 is recommended in infants receiving oxygenSaugstad OD. Oxygenation of the Immature Infant: A Commentary and Recommendations for Oxygen Saturation Targets and Alarm Limits. Neonatology, 2018;114:69–75.DOI: 10.1159/000486751 '''6.''' Bacterial contamination associated with certain nebulization and humidification systems is a possible hazardLa Fauci V, Costa GB, Facciolà A, Conti A, Riso R, Squeri R. Humidifiers for oxygen therapy: what risk for reusable and disposable devices? Journal of Preventive Medicine and Hygiene, 2017; 58: E161-E165. '''7.''' Oxygen therapy is contrindicated in patients suffering from [https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3243009/ paraquat poisoning]Gawarammana IB, Buckley NA. Medical management of paraquat ingestion. British Journal of clinical pharmacology, 2011; 72(5): 745–757. doi: 10.1111/j.1365-2125.2011.04026.x '''8. Pulmonary toxicity:''' Patients exposed to high oxygen levels for a prolonged period of time have lung damageMach WJ, Thimmesch AR, Pierce JT, Pierce JD. Consequences of Hyperoxia and the Toxicity of Oxygen in the Lung. Nursing Research and Practice, 2011. http://dx.doi.org/10.1155/2011/260482. The extent of lung damage is dependent on FiO2 and duration of exposure. It is due to intracellular free radicals (such as superoxide, activated hydroxyl ions, singlet O2 and hydrogen peroxide) formed which can damage alveolar-capillary membrane. It starts with increased permeability of the capillaries with resultant edema, thickened membranes and finally to pulmonary fibrosis. Care should be taken in the use of oxygen in patients receiving bleomycin or have previously used bleomycin Debnath J. Supplemental oxygen therapy in bleomycin-induced pulmonary toxicity: REPLY. Medical Journal, Armed forces India, 2011; 67(2): 194–195. doi: 10.1016/S0377-1237(11)60037-3.

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